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Acta Neuropathol 113, 265-275. Rockenstein E, Ubhi K, Pham E, Michael S, Doppler E, Novak P, Inglis C, Mante M, Adame A, Alvarez XA, Moessler H, Masliah E (2011) Beneficial consequences of a neurotrophic peptidergic mixture persist for a extended period pursuing therapy interruption in a transgenic product of Alzheimer’s illness. Int J Neuropsychopharmacol 12, 867-872. Alvarez XA, Cacabelos R, Sampedro C, Aleixandre M, Linares C, Granizo E, Doppler E, Moessler H (2011) Efficacy and safety of Cerebrolysin in moderate to moderately serious Alzheimer’s sickness: Results of a randomized, double-blind, managed trial investigating a few dosages of Cerebrolysin. Drugs Today (Barc) 48 Suppl A, 25-41. Alvarez XA, Cacabelos R, Sampedro C, Couceiro V, Aleixandre M, Vargas M, Linares C, Granizo E, GarciaFantini M, Baurecht W, Doppler E, Moessler H (2011) Combination procedure in Alzheimer’s condition: Results of a randomized, controlled demo with cerebrolysin and donepezil. J Neural Transm 112, 415-428. Alvarez XA, Cacabelos R, Laredo M, Couceiro V, Sampedro C, Varela M, Corzo L, Fernandez-Novoa L, Vargas M, Aleixandre M, Linares C, Granizo E, Muresanu D, Moessler H (2006) A 24-week, double-blind, placebocontrolled study of three dosages of Cerebrolysin in people with mild to reasonable Alzheimer’s illness. J Neural Transm Suppl 59, 293-300. Ruether E, Alvarez XA, Rainer M, Moessler H (2002) Sustained improvement of cognition and international purpose in sufferers with reasonably intense Alzheimer’s illness: A double-blind, placebo-managed examine with the neurotrophic agent Cerebrolysin.



J Neural Transm 112, 269282. Rockenstein E, Torrance M, Mante M, Adame A, Paulino A, Rose JB, Crews L, Moessler H, Masliah E (2006) Cerebrolysin decreases amyloid-beta production by regulating amyloid protein precursor maturation in a transgenic model of Alzheimer’s disorder. Matsuoka Y, Gray AJ, Hirata-Fukae C, Minami SS, Waterhouse EG, Mattson MP, LaFerla FM, Gozes I, Aisen PS (2007) Intranasal NAP administration cuts down accumulation of amyloid peptide and tau hyperphosphorylation in a transgenic mouse product of Alzheimer’s illness at early pathological stage. Peptides 31, 1368-1373. Fleming SM, Mulligan CK, Richter F, Mortazavi F, Lemesre V, Frias C, Zhu C, chaturbate-Token-to-dollar Stewart A, Gozes I, Morimoto B, Chesselet MF (2011) A pilot demo of the microtubule-interacting peptide (NAP) in mice overexpressing alpha-synuclein reveals improvement in motor functionality and reduction of alpha-synuclein inclusions. Neurosci Lett 361, 128-131. Gozes I, Zaltzman R, Hauser J, Brenneman DE, Shohami E, Hill JM (2005) The expression of action-dependent neuroprotective protein (ADNP) is controlled by mind harm and cure of mice with the ADNP derived peptide, NAP, reduces the severity of traumatic head harm. Drugs Today (Barc)48 Suppl A, 63-69. Leker RR, Teichner A, Grigoriadis N, Ovadia H, Brenneman DE, Fridkin M, Giladi E, Romano J, Gozes I (2002) NAP, a femtomolar-acting peptide, safeguards the brain versus ischemic injuries by cutting down apoptotic demise.



J Pharmacol Exp Ther 325, 146-153. Shiryaev N, Jouroukhin Y, Giladi E, Polyzoidou E, Grigoriadis NC, Rosenmann H, Gozes I (2009) NAP protects memory, improves soluble tau and reduces tau hyperphosphorylation in a tauopathy model. J Neural Transm Suppl 53, 313-321. Hutter-Paier B, Fruhwirth M, Grygar E, Windisch M (1996) Cerebrolysin guards neurons from ischemiainduced reduction of microtubule-connected protein 2. J Neural Transm Suppl 47, 276. Rockenstein E, Mallory M, Mante M, Alford M, Windisch M, Moessler H, Masliah E (2002) Effects of Cerebrolysin on amyloid-beta deposition in a transgenic product of Alzheimer’s condition. Curr Alzheimer Res 8, 583-591. Anton Alvarez X, Fuentes P (2011) Cerebrolysin in Alzheimer’s disease. Curr Alzheimer Res 4, 507-509. Merenlender-Wagner A, Pikman R, Giladi E, Andrieux A, Gozes I (2010) NAP (davunetide) boosts cognitive behavior in the Stop heterozygous mouse-a microtubuledeficient design of schizophrenia. Curr Alzheimer Res 2, 149-153. W. Froestl et al. Neurotox Res 7, 95-101. Rainer M, Brunnbauer M, Dunky A, Ender F, Goldsteiner H, Holl O, Kotlan P, Paulitsch G, Reiner C, Stossl J, Zachhuber C, Mossler H (1997) Therapeutic success with Cerebrolysin in the therapy of dementia. Drugs Aging 26, 893-915. Plosker GL, Gauthier S (2010) Spotlight on cerebrolysin in dementia.



Drugs Today (Barc) 48(Suppl A), 3-24. Panisset M, Gauthier S, Moessler H, Windisch M (2002) Cerebrolysin in Alzheimer’s ailment: A randomized, double-blind, placebo-managed trial with a neurotrophic agent. J Neurol Sci 299, 179-183. Plosker GL, Gauthier S (2009) Cerebrolysin: A critique of its use in dementia. J Neurol Sci 267, 112-119. Muresanu DF, Alvarez XA, Moessler H, Novak PH, Stan A, Buzoianu A, Bajenaru O, Popescu BO (2010) Persistence of the results of Cerebrolysin on cognition and qEEG slowing in vascular dementia clients: Results of a 3-thirty day period extension study. J Med Life 3, 137-143. Guekht AB, Moessler H, Novak PH, Gusev EI (2011) Cerebrolysin in vascular dementia: Improvement of clinical final result in a randomized, double-blind, placebocontrolled multicenter demo. Wien Med Wochenschr 147, 426-431. Xiong H, Baskys A, Wojtowicz JM (1996) Brain-derived peptides inhibit synaptic transmission by using presynaptic GABAB receptors in CA1 location of rat hippocampal slices. Arch Gerontol Geriatr 10, 129-140. Hutter-Paier B, Eggenreich U, Windisch M (1996) Effects of two protein-absolutely free peptide derivatives on passive avoidance behaviour of 24-thirty day period-outdated rats.